mouse lipocalin (R&D Systems)
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mouse lipocalin
Mouse Lipocalin, supplied by R&D Systems, used in various techniques. Bioz Stars score: 95/100, based on 149 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/ngal/Mouse+Lipocalin-2%2FNGAL+Quantikine+ELISA+Kit/pm41987381-41-19-23
Average 95 stars, based on 149 article reviews
Mouse Lipocalin, supplied by R&D Systems, used in various techniques. Bioz Stars score: 95/100, based on 149 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/ngal/Mouse+Lipocalin-2%2FNGAL+Quantikine+ELISA+Kit/pm41987381-41-19-23
Average 95 stars, based on 149 article reviews
mouse lipocalin - by Bioz Stars,
2026-09
95/100 stars
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Recombinant:Article Title: Development and provisional validation of a multiplex LC-MRM-MS test for timely kidney injury detection in urine Article Snippet: Recombinant SLC22A2 (Cat. Nr. H00006582-P01) was from Abnova (Taipei, Taiwan). .. Recombinant nephrin (Cat. Nr. 9399-NN-050) and biotinylated polyclonal antibodies against human TIMP2 (Cat. Nr. BAF971), IGFBP7 (Cat. Nr. BAF1334), KIM-1 (Cat. Nr. BAF1750), SDS Page:Article Title: Mac-1 Promotes Neutrophil Extracellular Traps Formation via ERK Phosphorylation in Renal Ischemia-Reperfusion Injury. Article Snippet: Proteins from kidney tissue, BMDN, or culture supernatant were extracted using radioimmunoprecipitation assay (RIPA) lysis buffer (20–188, Sigma-Aldrich, USA) supplemented with a protease and phosphatase inhibitor cocktail (P002, 1:100 dilutions, NCM Biotech, China). .. After SDS-PAGE and transfer to PVDF membranes, proteins were incubated overnight at 4 °C with primary antibodies, including Article Title: Pharmacological role of MLN4924 in cisplatin-induced acute kidney injury. Article Snippet: Background: Cisplatin-induced acute kidney injury (Cis-AKI) is a major complication that limits the clinical use of cisplatin, largely due to its cytotoxic effects on renal tubular epithelial cells.. Recent studies have shown that MLN4924, a NEDD8-activating enzyme inhibitor, can modulate key cellular processes such as apoptosis, autophagy, and DNA damage repair.. However, its precise role and regulatory mechanisms in the context of Cis-AKI remain largely undefined. Article Title: Mac-1 Promotes Neutrophil Extracellular Traps Formation via ERK Phosphorylation in Renal Ischemia-Reperfusion Injury Article Snippet: Proteins from kidney tissue, BMDN, or culture supernatant were extracted using radioimmunoprecipitation assay (RIPA) lysis buffer (20–188, Sigma-Aldrich, USA) supplemented with a protease and phosphatase inhibitor cocktail (P002, 1:100 dilutions, NCM Biotech, China). .. After SDS-PAGE and transfer to PVDF membranes, proteins were incubated overnight at 4 °C with primary antibodies, including Incubation:Article Title: Mac-1 Promotes Neutrophil Extracellular Traps Formation via ERK Phosphorylation in Renal Ischemia-Reperfusion Injury. Article Snippet: Proteins from kidney tissue, BMDN, or culture supernatant were extracted using radioimmunoprecipitation assay (RIPA) lysis buffer (20–188, Sigma-Aldrich, USA) supplemented with a protease and phosphatase inhibitor cocktail (P002, 1:100 dilutions, NCM Biotech, China). .. After SDS-PAGE and transfer to PVDF membranes, proteins were incubated overnight at 4 °C with primary antibodies, including Article Title: The role of FAK in renal collecting duct in the progression from acute kidney injury to chronic kidney disease. Article Snippet: AKI and CKD are major global health problem, which closely connected and promote each other.. The mechanism may be related to maladaptive repair after renal tubular injury.. FAK is a non-receptor tyrosine kinase located at the intersection of multiple cell signal transduction pathways. Article Title: DUSP26 protects against acute kidney injury by dephosphorylating p53 at serine 312. Article Snippet: .. After blocking with 5% non-fat milk, membranes were incubated overnight at 4°C with primary antibodies (all diluted at 1:1000) against DUSP26 (Invitrogen, PA5-22013), p53 (Cell Signaling Technology, 2524S), p-p53 (S15) (Cell Signaling Technology, 9284S), p-p53 (S20) (Cell Signaling Technology, 9287T), p-p53 (S46) (Proteintech, 28960), cleaved caspase-3 (Cell Signaling Technology, 9664S), Article Title: Pharmacological role of MLN4924 in cisplatin-induced acute kidney injury. Article Snippet: Background: Cisplatin-induced acute kidney injury (Cis-AKI) is a major complication that limits the clinical use of cisplatin, largely due to its cytotoxic effects on renal tubular epithelial cells.. Recent studies have shown that MLN4924, a NEDD8-activating enzyme inhibitor, can modulate key cellular processes such as apoptosis, autophagy, and DNA damage repair.. However, its precise role and regulatory mechanisms in the context of Cis-AKI remain largely undefined. Article Title: DUSP26 protects against acute kidney injury by dephosphorylating p53 at serine 312 Article Snippet: .. After blocking with 5% non-fat milk, membranes were incubated overnight at 4 °C with primary antibodies (all diluted at 1:1000) against DUSP26 (Invitrogen, PA5-22013), p53 (Cell Signaling Technology, 2524S), p-p53 (S15) (Cell Signaling Technology, 9284S), p-p53 (S20) (Cell Signaling Technology, 9287T), p-p53 (S46) (Proteintech, 28960), cleaved caspase-3 (Cell Signaling Technology, 9664S), Article Title: Mac-1 Promotes Neutrophil Extracellular Traps Formation via ERK Phosphorylation in Renal Ischemia-Reperfusion Injury Article Snippet: Proteins from kidney tissue, BMDN, or culture supernatant were extracted using radioimmunoprecipitation assay (RIPA) lysis buffer (20–188, Sigma-Aldrich, USA) supplemented with a protease and phosphatase inhibitor cocktail (P002, 1:100 dilutions, NCM Biotech, China). .. After SDS-PAGE and transfer to PVDF membranes, proteins were incubated overnight at 4 °C with primary antibodies, including Blocking Assay:Article Title: DUSP26 protects against acute kidney injury by dephosphorylating p53 at serine 312. Article Snippet: .. After blocking with 5% non-fat milk, membranes were incubated overnight at 4°C with primary antibodies (all diluted at 1:1000) against DUSP26 (Invitrogen, PA5-22013), p53 (Cell Signaling Technology, 2524S), p-p53 (S15) (Cell Signaling Technology, 9284S), p-p53 (S20) (Cell Signaling Technology, 9287T), p-p53 (S46) (Proteintech, 28960), cleaved caspase-3 (Cell Signaling Technology, 9664S), Article Title: Pharmacological role of MLN4924 in cisplatin-induced acute kidney injury. Article Snippet: Background: Cisplatin-induced acute kidney injury (Cis-AKI) is a major complication that limits the clinical use of cisplatin, largely due to its cytotoxic effects on renal tubular epithelial cells.. Recent studies have shown that MLN4924, a NEDD8-activating enzyme inhibitor, can modulate key cellular processes such as apoptosis, autophagy, and DNA damage repair.. However, its precise role and regulatory mechanisms in the context of Cis-AKI remain largely undefined. Article Title: DUSP26 protects against acute kidney injury by dephosphorylating p53 at serine 312 Article Snippet: .. After blocking with 5% non-fat milk, membranes were incubated overnight at 4 °C with primary antibodies (all diluted at 1:1000) against DUSP26 (Invitrogen, PA5-22013), p53 (Cell Signaling Technology, 2524S), p-p53 (S15) (Cell Signaling Technology, 9284S), p-p53 (S20) (Cell Signaling Technology, 9287T), p-p53 (S46) (Proteintech, 28960), cleaved caspase-3 (Cell Signaling Technology, 9664S), |
